NAD+ 100 | NAD100-X

XENE BIOLABS RESEARCH SERIES
FOR RESEARCH USE ONLY

Category: Coenzyme | Half-life: IV hours, SubQ 1-2 days

Mechanism

Essential coenzyme for ATP, DNA repair, sirtuins. Declines 50% by age 50. Direct NAD+ vs precursors NMN/NR.

Dosing Protocol

Reconstitution: 100 mg/mL. 50 mg = 0.5 mL = 50 units.

Critical: More isn’t better. 50 mg EOD beats 100 mg daily. Cycle 30 days on/off.

NAD-100X | Coenzyme / NAD+ Repletion | CAS: 53-84-9

NAD+ is a critical coenzyme in cellular energy metabolism, acting as an electron carrier in mitochondrial oxidative
phosphorylation. NAD+ levels decline with age, impacting mitochondrial function, sirtuin activity (SIRT1/3), DNA repair, and
metabolic regulation. IV or SubQ NAD+ directly replenishes cellular NAD+ with rapid systemic effect. Supports energy,
cognitive function, and all NAD+-dependent enzymatic processes.

DOSING PROTOCOL
ParameterDetails
IV Protocol250–1000 mg IV over 2–4 hours; weekly or monthly
SubQ25–50 mg SubQ daily (well-tolerated)
ReconstitutionSterile water; use immediately
CycleOngoing; no cycling required
STACKING RECOMMENDATIONS

Synergistic with 5-Amino-1MQ (NNMT inhibition raises endogenous NAD+), Epithalon (SIRT1/longevity), MOTS-c
(mitochondrial NAD+ utilization), Semax/Selank (neuroprotective NAD+ demand).

NOTE: IV administration causes transient flushing, chest tightness, and nausea — slow infusion rate. SubQ is better tolerated. NMN/NR
are oral NAD+ precursors that can supplement between IV sessions.